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| Pyosalpinx | ||
| Etiology •secondary to bacterial infection usually Chlamydia trachomatis or Neisseria Gonorrhae when sexually transmitted or •staphylococci, streptoccoci, or coliforms •organisms reach fallopian tube by ascending through cervix and across endometrium | ||
| Pathogenesis •suppurative acute inflammatory response to organisms within the fallopian tube •may occur rapidly following vaginal inoculation with organism (3-5 days in experimental models) •may be facilitated by estrogen •fusion of the end of the tube results in purulent accumulation within the dilated tube, | ||
| Epidemiology •part of Pelvic Inflammatory Disease (P.I.D.) which may be •sexually transmitted disease or •follow delivery, abortion, instrumentation, or IUD placement •disease of reproductive age women | ||
| General Gross Description •enlarged fallopian tube with hyperemic external surface which may show serositis •lumen filled with pus •Examples: | ||
| General Microscopic Description •acute inflammatory infiltrate within the lumen and involving epithelium of the tube •Examples: | ||
| References • Cotran RS, Kumar V, Robbins SL: Robbins Pathologic Basis of Disease. 5th ed. Philadelphia, W.B. Saunders, 1994, pp. 1039 This link will directly take you to the relevant new literature Pyosalpinx
| Synopsis by: Melinda Sanders M.D. (T86100M40460)[2]
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