| Acute Vasomotor Nephropathy
|
Etiology
•Inadequate blood flow to both kidneys (shock).
|
Pathogenesis
•The reduction
in blood flow is inadequate to maintain renal function and yet not complete
and sustained enough to cause infarction.
•The condition is reversible.,
|
Epidemiology
•Frequent complication
of shock.
|
General Gross Description
•The renal cortex is pale and the medulla is dark and congested.
•Examples:
|
General Microscopic Description
•The
tubular epithelium, especially of the proximal tubules undergoes necrosis and
sloughs into the tubular lumens. These cells degenerate into amorphous
clumps sometimes seen in more distal portions of the nephron as granular
pigmented casts.
•The vasa recta are congested and accumulate increased
numbers of white cells.
•Examples:
|
Clinical Correlation
•Acute renal failure.
|
References
• Cotran RS, Kumar V, Robbins SL: Robbins Pathologic Basis of Disease. 5th ed. Philadelphia, W.B. Saunders, 1994, pp. 964-967.
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| Acute Vasomotor Nephropathy
| | Synopsis by: Harold Yamase M.D. (T71000M53150)[184]
| |